By A.H. Rose, D.W. Tempest (Eds.)
This quantity in a research-level sequence covers assorted elements of microbial body structure and biochemistry together with inositol metabolisms in yeasts, bacterial adhesion, natural acids, the bacterial flagellum and the mechanical behaviour of bacterial cellphone partitions. it's meant to be of use to microbiologists, biochemists and biotechnologists. different comparable works during this sequence are volumes 29, 30 and 31.
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Additional resources for Advances in Microbial Physiology, Vol. 28
R. WOODS AND D. T. JONES The B. 6 at 1% survival, as compared with the wild type. The main difference between the UV survival curves of the wild-type and UVS9 strains was in the shoulder region where repair of the DNA damage occurs at low doses of UV radiation. With E. coli, liquid-holding recovery is observed in cells having functional uur andpolA gene products, but only if they have some other decreased repair capacity as is found in E. coli K12 or E. frugilis, aerobic liquid-holding recovery occurs in the wild type as well as in both mutant strains.
The development and use of defined media for the growth and sporulation of C. botulinum was reported by Perkinsand Tsuji (1962) for type A strains, and by Ward and Carroll (1966) and Strasdine and Melville (1968) for type E strains. Owing to the complex nutritional requirementsof many of the clostridia the synthetic media described thus far all contain most of the commonly occurring 30 D. R. WOODS AND D. T. JONES amino acids and an unwieldy supply of vitamins and minerals. Less complicated minimal media have been developed for the saccharolytic clostridia where the amino acids may be replaced by a single inorganic nitrogen source.
Although, as an obligate anaerobe, B. , 1982). Proteins with similar molecular weights are induced by 0 2 and H202, but not by heat. Ultraviolet irradiation, 0 2 and H202 induce phage reactivation systems but heat stress does not. Since W irradiation, 0 2 and H202 cause DNA damage and induce DNA repair mechanisms, it is suggested that the physiological responses to these stress factors have aspects in common with, but differ from, the heat-shock response. A further difference, suggesting different control systems, involved the rate of protein induction.
Advances in Microbial Physiology, Vol. 28 by A.H. Rose, D.W. Tempest (Eds.)